Helpful hint for your Diabetes Claim

Discussion in 'Diabetes' started by Rougetet, Apr 27, 2008.

  1. Rougetet

    Rougetet New Member

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    While recently doing some research for my service-connected Diabetes Mellitus (DM), I reviewed all Board of Veteran Appeals (BVA) decisions in 2007 and 2008 (first four months) concerning requests to increase a rating from 20% to 40% for DM and noticed what appeared to be a “catch-22” situation that often resulted in a denial by the BVA for the increase. DM at the 40% rating level requires that there be evidence of a medical finding of “regulation of activities” in the medical record. If the veteran has been encouraged to exercise or is admittedly exercising for assistance with DM control, that has been found by the BVA to be evidence that there is no regulation of activities absent any specific medical notes of regulation of activity restrictions. Since habitually medical providers will encourage diabetics to exercise, in many cases as a physical therapy to any obesity, this quite often sets up an “automatic” denial for the increase requested. I support exercising as a means of assisting DM control, but don’t believe it should be in of itself the cause for any increase denial if no other regulation of activities wording shown in the medical record. Additionally, ALL C&P examinations for DM are requested to provide indication of any regulation of activities requirements that are in the medical record. If no VA or private physician has indicated specifically that there were regulation of activities, the C&P examiner will include a statement that there was no regulation of activities present in their C&P findings (near as I can tell their guidelines do not allow them to make a medical determination there was regulation of activities without one previously being in the claim file – they are only reporting what they find in the claim file as regards regulation of activities in a C&P exam). Of the 336 DM cases found in my 2007-2008 BVA case research for veterans that had a 20% DM rating and were seeking an increase and case not remanded by the BVA, denial because the 40% rating requirement for regulation of activities was not met as the primary denial reason occurred in 60.4% of these cases.

    In order to counteract this somewhat automatic denial issue for veterans with service-connected DM, consider printing out the next two paragraphs on a sheet of paper and providing this to your VA primary care physician on your next visit:

    Can you agree to advise me on the statement in the next paragraph and include the statement in that paragraph exactly as written in your medical notes for this visit? If not, what can you agree to place in your notes for this visit that would be close to the wording as shown?

    “The patient was advised to avoid strenuous occupational and recreation activities that would either raise their stress to levels that would adversely affect their blood sugars or increase their frequency of hypoglycemic episodes.”

    Also prior to the visit with your VA primary care physician, print out the ADA’s take on stress and diabetes and show that to the physician in case the physician disagrees with the stress part of the statement. That page can be found on the Internet here: http://www.diabetes.org/type-2-diabetes/stress.jsp

    If the VA primary care physician wants to revise the statement, the most important words to be sure are included are “avoid strenuous occupational and recreational activities”.

    If your VA primary care physician will not agree to provide a statement substantially similar to the previous paragraph in their treatment notes, seek out a private care physician for a second opinion.

    For private care physicians where you are seeking a 2nd opinion, the wording should be revised to something similar to the following when asking them to provide supporting medical opinions in their care notes for the visit:

    “The patient came in today with their VA medical records to seek out a 2nd opinion on their diabetic care. After careful review of the medical history provided and questioning them on their specific diabetic condition and symptoms, I advised the patient to avoid strenuous occupational and recreation activities that would either raise their stress to levels that would adversely affect their blood sugars or increase their frequency of hypoglycemic episodes.”

    It is important to stress to the physician in either case that you are not asking them to lie about your condition, just asking them if they can medically support adding this statement to your medical records.
  2. will64

    will64 New Member

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    Proposed Medical Entry

    Rouget - I think your hint provides the last piece I need to have a decent chance on having my rating for DM increased from 20 to 40%! I think my physician might agree to the following wording:

    "The patient has been advised to avoid strenuous occupational and recreational activities to prevent any increase in the severity or frequency of low blood sugar/hypoglycemica. Activities should be limited to light and moderate activities with blood glucose management measures in accordance with ADA guidelines."

    Do you think this statement is good enough? And do you think the light to moderate comment clouds things too much?

    Another question. In your review of appeal cases, did you notice any problem getting VA to accepting adding insulin as medically necessary vs. just improved control? My situation is pretty clear - 4 oral meds plus Byetta wasn't effective so my only choice was insulin.

    will64
  3. CEDAR FALLS

    CEDAR FALLS Member

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    Neuropaty with diabetes mental disease ???

    I dont see mental disease mentioned ??

    Peripheral neuropathyFrom Wikipedia, the free encyclopedia
    Jump to: navigation, search
    "Neuropathy" redirects here. For other uses, see Neuropathy (disambiguation).
    Not to be confused with Nephropathy.
    Peripheral neuropathy
    Classification and external resources
    ICD-10 G64., G90.0
    ICD-9 356.0, 356.8
    DiseasesDB 9850
    MeSH D010523
    Peripheral neuropathy is the term for damage to nerves of the peripheral nervous system,[1] which may be caused either by diseases of or trauma to the nerve or the side-effects of systemic illness.

    The four cardinal patterns of peripheral neuropathy are polyneuropathy, mononeuropathy, mononeuritis multiplex and autonomic neuropathy. The most common form is (symmetrical) peripheral polyneuropathy, which mainly affects the feet and legs. The form of neuropathy may be further broken down by cause, or the size of predominant fiber involvement, i.e., large fiber or small fiber peripheral neuropathy. Frequently the cause of a neuropathy cannot be identified and it is designated idiopathic.

    Neuropathy may be associated with varying combinations of weakness, autonomic changes, and sensory changes. Loss of muscle bulk or fasciculations, a particular fine twitching of muscle, may be seen. Sensory symptoms encompass loss of sensation and "positive" phenomena including pain. Symptoms depend on the type of nerves affected (motor, sensory, or autonomic) and where the nerves are located in the body. One or more types of nerves may be affected. Common symptoms associated with damage to the motor nerve are muscle weakness, cramps, and spasms. Loss of balance and coordination may also occur. Damage to the sensory nerve can produce tingling, numbness, and pain. Pain associated with this nerve is described in various ways such as the following: sensation of wearing an invisible "glove" or "sock", burning, freezing, or electric-like, extreme sensitivity to touch. The autonomic nerve damage causes problems with involuntary functions leading to symptoms such as abnormal blood pressure and heart rate, reduced ability to perspire, constipation, bladder dysfunction (e.g., incontinence), and sexual dysfunction.[2]

    Contents [hide]
    1 Classification
    1.1 Mononeuropathy
    1.2 Mononeuritis multiplex
    1.3 Polyneuropathy
    1.4 Autonomic neuropathy
    1.5 Neuritis
    2 Signs and symptoms
    3 Causes
    4 Treatment
    5 References
    6 External links

    [edit] ClassificationPeripheral neuropathy may be classified according to the number of nerves affected or the type of nerve cell affected (motor, sensory, autonomic), or the process affecting the nerves (e.g. inflammation in neuritis).

    [edit] MononeuropathySee also: Compression neuropathy
    Mononeuropathy is a type of neuropathy that only affects a single nerve.[3] It is diagnostically useful to distinguish them from polyneuropathies, because the limitation in scope makes it more likely that the cause is a localized trauma or infection.

    The most common cause of mononeuropathy is by physical compression of the nerve, known as compression neuropathy. Carpal tunnel syndrome is one example of this. The "pins-and-needles" sensation of one's "foot falling asleep" (paresthesia) is caused by a compression mononeuropathy, albeit a temporary one which can be resolved merely by moving around and adjusting to a more appropriate position. Direct injury to a nerve, interruption of its blood supply (ischemia), or inflammation can also cause mononeuropathy.

    [edit] Mononeuritis multiplexMononeuritis multiplex is simultaneous or sequential involvement of individual noncontiguous nerve trunks, either partially or completely, evolving over days to years and typically presents with acute or subacute loss of sensory and motor function of individual peripheral nerves. The pattern of involvement is asymmetric, however, as the disease progresses, deficit(s) becomes more confluent and symmetrical, making it difficult to differentiate from polyneuropathy. Therefore, attention to the pattern of early symptoms is important.

    Mononeuritis multiplex may also cause pain, which is characterized as deep, aching pain that is worse at night, is frequently in the lower back, hip, or leg. In people with diabetes mellitus, mononeuritis multiplex is typically encountered as acute, unilateral, severe thigh pain followed by anterior muscle weakness and loss of knee reflex.

    Electrodiagnostic studies will show multifocal sensory motor axonal neuropathy.

    It is caused by, or associated with, several medical conditions:

    diabetes mellitus
    vasculitides: polyarteritis nodosa, Wegener granulomatosis, and Churg-Strauss syndrome
    immune-mediated diseases like rheumatoid arthritis, lupus erythematosus (SLE), and sarcoidosis
    infections: leprosy, lyme disease, HIV
    amyloidosis
    cryoglobulinemia
    chemical agents, including trichloroethylene and dapsone
    [edit] PolyneuropathyMain article: Polyneuropathy
    Polyneuropathy is a pattern of nerve damage which is quite different from mononeuropathy. The term "peripheral neuropathy" is sometimes used loosely to refer to polyneuropathy. In a polyneuropathy, many nerve cells in different parts of the body are affected, without regard to the nerve through which they pass. Not all nerve cells are affected in any particular case. In distal axonopathy, one common pattern, the cell bodies of neurons remain intact, but the axons are affected in proportion to their length. Diabetic neuropathy is the most common cause of this pattern. In demyelinating polyneuropathies, the myelin sheath around axons is damaged, which affects the ability of the axons to conduct electrical impulses. The third and least common pattern affects the cell bodies of neurones directly. This usually picks out either the motor neurones (known as motor neurone disease) or the sensory neurones (known as sensory neuronopathy or dorsal root ganglionopathy).

    The effect of this is to cause symptoms in more than one part of the body, often on left and right sides symmetrically. As for any neuropathy, the chief symptoms include weakness or clumsiness of movement (motor); unusual or unpleasant sensations such as tingling or burning; reduction in the ability to feel texture, temperature, etc.; and impaired balance when standing or walking (sensory). In many polyneuropathies, these symptoms occur first and most severely in the feet. Autonomic symptoms may also occur, such as dizziness on standing up, erectile dysfunction and difficulty controlling urination.

    Polyneuropathies are usually caused by processes that affect the body as a whole. Diabetes and impaired glucose tolerance are the most common causes. Other causes relate to the particular type of polyneuropathy, and there are many different causes of each type, including inflammatory diseases such as lyme disease, vitamin deficiencies, blood disorders, and toxins (including alcohol and certain prescribed drugs). Most types of polyneuropathy progress fairly slowly, over months or years, but rapidly progressive polyneuropathy also occurs. It is important to recognize that glucose levels in the blood can spike to nerve-damaging levels after eating even though fasting blood sugar levels and average blood glucose levels can still remain below normal levels (currently typically considered below 100 for fasting blood plasma and 6.0 for HGBA1c, the test commonly used to measure average blood glucose levels over an extended period). Studies have shown that many of the cases of peripheral small fiber neuropathy with typical symptoms of tingling, pain and loss of sensation in the feet and hands are due to glucose intolerance before a diagnosis of diabetes or pre-diabetes. Such damage is often reversible, particularly in the early stages, with diet, exercise and weight loss.17

    The treatment of polyneuropathies is aimed firstly at eliminating or controlling the cause, secondly at maintaining muscle strength and physical function, and thirdly at controlling symptoms such as neuropathic pain.

    [edit] Autonomic neuropathyAutonomic neuropathy is a form of polyneuropathy which affects the non-voluntary, non-sensory nervous system (i.e., the autonomic nervous system) affecting mostly the internal organs such as the bladder muscles, the cardiovascular system, the digestive tract, and the genital organs. These nerves are not under a person's conscious control and function automatically. Autonomic nerve fibers form large collections in the thorax, abdomen and pelvis outside spinal cord, however they have connections with the spinal cord and ultimately the brain. Most commonly autonomic neuropathy is seen in persons with long-standing diabetes mellitus type 1 and 2. In most but not all cases, autonomic neuropathy occurs alongside other forms of neuropathy, such as sensory neuropathy.

    Autonomic neuropathy is one cause of malfunction of the autonomic nervous system, but not the only one; some conditions affecting the brain or spinal cord can also cause autonomic dysfunction, such as multiple system atrophy, and therefore cause similar symptoms to autonomic neuropathy.

    The signs and symptoms of autonomic neuropathy include the following:

    urinary bladder conditions: bladder incontinence or urine retention
    gastrointestinal tract: dysphagia, abdominal pain, nausea, vomiting, malabsorption, fecal incontinence, gastroparesis, diarrhea, constipation
    cardiovascular system: disturbances of heart rate (tachycardia, bradycardia), orthostatic hypotension, inadequate increase of heart rate on exertion
    other: hypoglycemia unawareness, genital impotence, sweat disturbances
    [edit] NeuritisNeuritis is a general term for inflammation of a nerve[4] or the general inflammation of the peripheral nervous system. Symptoms depend on the nerves involved, but may include pain, paresthesia (pins & needles), paresis (weakness), hypoesthesia (numbness), anesthesia, paralysis, wasting, and disappearance of the reflexes. Causes include:




    Physical injury
    One common cause of neuritis and subsequent inflammation of the nerves to the toes is the wearing of high-heeled shoes or ill-fitting shoes that bind the toes painfully. This can cause temporary numbness and pain in the affected toes for several days. An effective treatment is Neuromethyn (also known as methylcobalamin or mecobalantin) which can be prescribed by a doctor.

    Infection:
    Herpes simplex
    Shingles
    Leprosy
    Guillain-Barre syndrome
    Lyme Disease



    Chemical injury
    Radiation
    Underlying conditions causing localized neuritis (affecting a single nerve):
    Diphtheria
    Localized injury
    Diabetes
    Underlying conditions causing polyneuritis (affecting multiple nerves):
    Beriberi
    Vitamin B12 deficiency
    Metabolic diseases
    Diabetes
    Hypothyroidism
    Porphyria
    Infections, bacterial and/or viral
    Autoimmune disease, especially Multiple Sclerosis
    Cancer
    Alcoholism
    Wartenbergs migratory sensory neuropathy
    Types of neuritis include:

    Polyneuritis or Multiple neuritis (not to be confused with multiple sclerosis)
    Brachial neuritis
    Optic neuritis
    Vestibular neuritis
    Cranial neuritis, often representing as Bell's Palsy
    Arsenic neuritis
    [edit] Signs and symptomsThose with diseases or dysfunctions of their peripheral nerves can present with problems in any of the normal peripheral nerve functions.

    In terms of sensory function, there are commonly loss of function (negative) symptoms, which include numbness, tremor, and gait abnormality.

    Gain of function (positive) symptoms include tingling, pain, itching, crawling, and pins and needles. Pain can become intense enough to require use of opioid (narcotic) drugs (i.e., morphine, oxycodone).

    Skin can become so hypersensitive that patients are prohibited from having anything touch certain parts of their body, especially the feet. People with this degree of sensitivity cannot have a bedsheet touch their feet or wear socks or shoes, and eventually become housebound.

    Motor symptoms include loss of function (negative) symptoms of weakness, tiredness, heaviness, and gait abnormalities; and gain of function (positive) symptoms of cramps, tremor, and muscle twitch (fasciculations).

    There is also pain in the muscles (myalgia), cramps, etc., and there may also be autonomic dysfunction.

    During physical examination, those with generalized peripheral neuropathies most commonly have distal sensory or motor and sensory loss, though those with a pathology (problem) of the peripheral nerves may be perfectly normal; may show proximal weakness, as in some inflammatory neuropathies like Guillain–Barré syndrome; or may show focal sensory disturbance or weakness, such as in mononeuropathies. Ankle jerk reflex is classically absent in peripheral neuropathy.

    [edit] CausesThe causes are broadly grouped as follows:

    Genetic diseases: Friedreich's ataxia, Charcot-Marie-Tooth syndrome [5]
    Metabolic/Endocrine: diabetes mellitus [6], chronic renal failure, porphyria, amyloidosis, liver failure, hypothyroidism
    Toxic causes: Drugs (vincristine, phenytoin, nitrofurantoin, isoniazid, ethyl alcohol), organic metals, heavy metals, excess intake of vitamin B6 (pyridoxine)
    Fluoroquinolone toxicity: Irreversible neuropathy is a serious adverse reaction of fluoroquinolone drugs [7]
    Inflammatory diseases: Guillain-Barré syndrome, systemic lupus erythematosis, leprosy, Sjögren's syndrome, Lyme Disease,sarcoidosis,[8]
    Vitamin deficiency states: Vitamin B12 (cyanocobalamin), vitamin A, vitamin E, vitamin B1 (thiamin)
    Physical trauma: compression, pinching, cutting, projectile injuries (i.e. gunshot wound), strokes including prolonged occlusion of blood flow
    Others: shingles, malignant disease, HIV [9], radiation, chemotherapy[10]
    Many of the diseases of the peripheral nervous system may present similarly to muscle problems (myopathies), and so it is important to develop approaches for assessing sensory and motor disturbances in patients so that a physician may make an accurate diagnosis.

    [edit] TreatmentMany treatment strategies for peripheral neuropathy are symptomatic. Some current research in animal models has shown that neurotrophin-3 can oppose the demyelination present in some peripheral neuropathies.[11]

    A range of drugs that act on the central nervous system such as drugs originally intended as antidepressants and antiepileptic drugs have been found to be useful in managing neuropathic pain. Commonly used treatments include using a tricyclic antidepressant (such as amitriptyline) and antiepileptic therapies such as gabapentin or sodium valproate. These have the advantage that besides being effective in many cases they are relatively low cost.

    A great deal of research has been done between 2005 and 2010 which indicates that synthetic cannabinoids and inhaled cannabis are effective treatments for a range of neuropathic disorders. [12] Research has demonstrated that the synthetic oral cannabinoid Nabilone is an effective adjunct treatment option for neuropathic conditions, especially for people who are resistant, intolerant, or allergic to common medications.[13] Orally, opiate derivatives were found to be more effective than cannabis for most people.[14] Smoked cannabis has been found to provide relief from HIV-associated sensory neuropathy. [15] Smoked cannabis was also found to relieve neuropathy associated with CRPS type I, spinal cord injury, peripheral neuropathy, and nerve injury. [16]

    Pregabalin (INN, pronounced /prɨˈɡæbəlɨn/) is an anticonvulsant drug used for neuropathic pain. It has also been found effective for generalized anxiety disorder. It was designed as a more potent successor to gabapentin but is significantly more expensive, especially now that the patent on gabapentin has expired and gabapentin is available as a generic drug. Pregabalin is marketed by Pfizer under the trade name Lyrica.

    TENS (Transcutaneous Electrical Nerve Stimulation) therapy may be effective and safe in the treatment of diabetic peripheral neuropathy. A recent review of three trials involving 78 patients found some improvement in pain scores after 4 and 6 but not 12 weeks of treatment, and an overall improvement in neuropathic symptoms at 12 weeks.[17] A second review of four trials found significant improvement in pain and overall symptoms, with 38% of patients in one trial becoming asymptomatic. The treatment remains effective even after prolonged use, but symptoms return to baseline within a month of treatment cessation.[18]

    [edit] References^ "Peripheral Neuropathy Fact Sheet: National Institute of Neurological Disorders and Stroke (NINDS)". Peripheral Neuropathy Fact Sheet: National Institute of Neurological Disorders and Stroke (NINDS). Retrieved 2008-11-30.
    ^ Neuropathy Signs & Symptoms - Neuropathy - Neurology Channel
    ^ "Dorlands Medical Dictionary:mononeuropathy". http://www.mercksource.com/pp/us/cn...us/common/dorlands/dorland/five/000067367.htm.
    ^ neuritis at Dorland's Medical Dictionary
    ^ Gabriel JM, Erne B, Pareyson D, Sghirlanzoni A, Taroni F, Steck AJ (1997). "Gene dosage effects in hereditary peripheral neuropathy. Expression of peripheral myelin protein 22 in Charcot-Marie-Tooth disease type 1A and hereditary neuropathy with liability to pressure palsies nerve biopsies". Neurology 49 (6): 1635–40. PMID 9409359.
    ^ Kiziltan ME, Akalin MA, Sahin R, Uluduz D (2007). "Peripheral neuropathy in patients with diabetes mellitus presenting as Bell's palsy". Neuroscience Letters 427 (3): 138. doi:10.1016/j.neulet.2007.09.029. PMID 17933462.
    ^ Cohen JS (December 2001). "Peripheral Neuropathy Associated with Fluoroquinolones" (PDF). Ann Pharmacother 35 (12): 1540–7. doi:10.1345/aph.1Z429. PMID 11793615. http://fqvictims.org/fqvictims/News/neuropathy/Neuropathy.pdf.
    ^ Heck AW, Phillips LH 2nd (1989). "Sarcoidosis and the nervous system". Neurol Clin 7 (3): 641–54. PMID 2671639.
    ^ Gonzalez-Duarte A, Cikurel K, Simpson DM (2007). "Managing HIV peripheral neuropathy". Current HIV/AIDS reports 4 (3): 114–8. doi:10.1007/s11904-007-0017-6. PMID 17883996.
    ^ Wilkes G (2007). "Peripheral neuropathy related to chemotherapy". Seminars in oncology nursing 23 (3): 162–73. doi:10.1016/j.soncn.2007.05.001. PMID 17693343.
    ^ Liu N, Varma S, Tsao D, Shooter EM, Tolwani RJ (2007). "Depleting endogenous neurotrophin-3 enhances myelin formation in the Trembler-J mouse, a model of a peripheral neuropathy". J. Neurosci. Res. 85 (13): 2863–9. doi:10.1002/jnr.21388. PMID 17628499.
    ^ http://www.cannabis-med.org/data/pdf/en_2010_01_special.pdf
    ^ Skrabek RQ, Galimova L, Ethans K, Perry D (2008). "Nabilone for the treatment of pain in fibromyalgia". J. Pain 9 (2): 164–73. doi:10.1016/j.jpain.2007.09.002. PMID 17974490.
    ^ Frank B, Serpell MG, Hughes J, Matthews JN, Kapur D (2008). "Comparison of analgesic effects and patient toleration of nabilone and dihydrocodeine for chronic neuropathic pain: randomized, crossover, double blind study". BMJ 336 (7637): 119–201.
    ^ Abrams DI, Jay CA, Shade SB, Vizozo H, Reda H, Press S, Kelly ME, Rowbotham Mc, Petersen KL (2007). "Cannabis in painful HIV-associated sensory neuropathy: a randomized placebo-controlled trail". J. Neurology 68 (7): 515–21. doi:10.1212/01.wnl.0000253187.66183.9c. PMID 17296917.
    ^ Wilsey B, Marcotte T, Tsodikov A, Millman J, Bentley H, Gouaux B, Fishman S (2008). "A randomized, placebo-controlled, crossover trail of cannabis cigarettes in neuropathic pain". J. Pain 9 (6): 506–21. doi:10.1016/j.jpain.2007.12.010. PMID 18403272.
    ^ Jin DM, Xu Y, Geng DF, Yan TB (July 2010). "Effect of transcutaneous electrical nerve stimulation on symptomatic diabetic peripheral neuropathy: a meta-analysis of randomized controlled trials". Diabetes Res. Clin. Pract. 89 (1): 10–5. doi:10.1016/j.diabres.2010.03.021. PMID 20510476.
    ^ Pieber K, Herceg M, Paternostro-Sluga T (April
  4. abc160561b1

    abc160561b1 Member

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